p53蛋白在骨性关节炎中的作用机制及研究进展
The mechanism and research progress of p53 protein in osteoarthritis
  
DOI:10.3969/j.issn.1006-7108.2026.05.021
中文关键词:  p53  炎症因子  铁死亡  软骨细胞  自噬  信号通路
英文关键词:p53  inflammatory factors  ferroptosis  chondrocytes  autophagy  signaling pathways
基金项目:甘肃省自然科学基金(22JR11RA197);甘肃省临床医学研究中心建设项目(18JR2FA009)
作者单位
杨昊飞1 李爱强2* 朱旭东1 张旭升1 李金生1 柳海平2 1.甘肃中医药大学,甘肃 兰州 730000 2.甘肃省中医院,甘肃兰州 730050 
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中文摘要:
      骨性关节炎(osteoarthritis,OA)是一种常见于中老年人群的骨代谢疾病,主要以关节软骨损伤、骨微结构破坏、炎症反应的产生为特征,临床表现以疼痛、肿胀、僵硬、活动受限为主。OA早期以物理治疗、药物干预等治疗为主,存在多药耐药、不良反应多等局限性。随着OA机制的不断深入研究,相关调控因子陆续被发现,p53蛋白是细胞内重要的调控因子,发现其调节着OA中软骨细胞的增殖、分化、凋亡、自噬等过程,表明p53的表达对骨的功能及代谢有重要的调节作用。本文通过p53介导炎症因子、软骨细胞、炎症信号通路、铁死亡等机制探讨OA的发病机制,p53将成为治疗OA等骨代谢疾病的新靶点。
英文摘要:
      Osteoarthritis (OA) is a bone metabolic disease commonly found in middle-aged and elderly people. It is mainly characterized by joint cartilage injury, bone microstructure destruction, and inflammatory response. The clinical manifestations are mainly pain, swelling, stiffness, and limited activity. In the early stage of OA, physical therapy and drug intervention are the main treatments, and there are many limitations such as multi-drug resistance and adverse reactions. With the continuous in-depth study of the mechanism of OA, relevant regulatory factors have been discovered one after another. p53 protein is an important intracellular regulatory factor, which is found to regulate the proliferation, differentiation, apoptosis, autophagy and other processes of chondrocytes in OA, indicating that the expression of p53 plays an important regulatory role in bone function and metabolism. In this paper, the pathogenesis of OA was explored through p53 mediating inflammatory factors, chondrocytes, inflammatory signaling pathways, iron death and other mechanisms, and p53 will become a new target for the treatment of bone metabolic diseases such as OA.
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